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Sustanon was originally designed for HRT (hormone replacement therapy), so the 4 testosterones would allow sustanon to stay in your system for up to 4 weeks. This is the reason that the 4 testosterones are called “maintenance” hormones and not anabolic hormones, i.e. the progestational hormone progesterone.
It was later discovered that one of the primary functions of sustanon (and progesterone) was to prevent tumor cell formation by stimulating the secretion of the protein tumor necrosis factor, but that function can sometimes be impaired by certain drugs, so the 4 testosterones are called “maintainers” of prostaglandin synthesis.
When progesterone is administered to the intact body (progestational estrogen), progesterone is converted to pregnenolone in some cells, and this is the primary source of estrogen in the body (although some progesterones may be metabolized by the liver, in particular prostaglandin E2).
The use of estrogens in humans (or animals) is quite rare, and the effect of estrogens on pregnancy is extremely small (the few experiments conducted actually had low effects, possibly due to the severe lack of control, or due to the fact that progesterone can suppress some important hormones as well, such as progesterone-binding globulin/glucuronide and thyroid hormones).
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In addition, progestin is an endogenous antagonist of progesterone receptors, and therefore cannot bind to these receptors on the prostate. That is to say, pregnenolone can only act in the human body when progesterone is present.
Another factor in the use of progesterone during the first trimester is that progesterone is metabolized by the liver and glucuronidation occurs with other drugs. But some progesterone is also created by the liver in response to progesterone. The liver (which produces progesterone) can oxidize prostaglandin E2 into prostaglandin B2, so prostaglandin E2 can be converted to a form of progesterone in tissues in which it is present
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